Acute Coronary Syndrome (ACS) is a term that used to describe the spectrum of circumstances or set of processes disease including unstable angina pectoris / UA (unstable angina / UA), myocardial infarction non-Q wave, or myocardial infarction without ST segment elevation (Non-ST elevation myocardial infarction / NSTEMI), and myocardial infarction or Q wave myocardial infarction with ST segment elevation (ST elevation myocardial infarction / STEMI.
UA and NSTEMI have pathogenesis and clinical presentation are similar, differing only in degree. If found biochemical markers of myocardial necrosis (elevated troponin I, troponin T, or CK-MB), the diagnosis was NSTEMI, whereas when the marker biochemistry is not raised, then the diagnosis is UA. At the UA and NSTEMI blood vessels involved did not have total occlusion / no total occlusions (patency), and so we need to prevent plaque stabilization progression, thrombosis and vasoconstriction. Determination of troponin I / T features the most sensitive and specific for myocyte necrosis and pathogenesis and sequence determination treatment. Infarction were still influenced by the need of medicines working towards the heart, the ultimate burden, inotropic state, the initial burden to reduce myocardial O2 consumption. UA and NSTEMI ACS is a characterized by an imbalance of supply and myocardial oxygen demand. The main causes of coronary stenosis due to non-occlusive thrombus occurring the atherosclerotic plaque erosion, fissure, or rupture.
The three types of coronary events was in fact a process of stages: from the phenomenon of light to the heaviest. And levels of the mainly influenced by collateralization, the level of occlusion, or absence of acute and duration of myocardial ischemia lasts. More discussion on this guide focused on the problem of the two parts of this syndrome: Angina unstable pectoris (UA) and NSTEMI.
UA and NSTEMI have pathogenesis and clinical presentation are similar, differing only in degree. If found biochemical markers of myocardial necrosis (elevated troponin I, troponin T, or CK-MB), the diagnosis was NSTEMI, whereas when the marker biochemistry is not raised, then the diagnosis is UA. At the UA and NSTEMI blood vessels involved did not have total occlusion / no total occlusions (patency), and so we need to prevent plaque stabilization progression, thrombosis and vasoconstriction. Determination of troponin I / T features the most sensitive and specific for myocyte necrosis and pathogenesis and sequence determination treatment. Infarction were still influenced by the need of medicines working towards the heart, the ultimate burden, inotropic state, the initial burden to reduce myocardial O2 consumption. UA and NSTEMI ACS is a characterized by an imbalance of supply and myocardial oxygen demand. The main causes of coronary stenosis due to non-occlusive thrombus occurring the atherosclerotic plaque erosion, fissure, or rupture.
The three types of coronary events was in fact a process of stages: from the phenomenon of light to the heaviest. And levels of the mainly influenced by collateralization, the level of occlusion, or absence of acute and duration of myocardial ischemia lasts. More discussion on this guide focused on the problem of the two parts of this syndrome: Angina unstable pectoris (UA) and NSTEMI.
